C/EBP homologous protein inhibits tissue repair in response to gut injury and is inversely regulated with chronic inflammation-Reference-Cited by-同舟云学术

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C/EBP homologous protein inhibits tissue repair in response to gut injury and is inversely regulated with chronic inflammation

Published:2014-05-21 Issue:6 Volume:7 Page:1452-1466
ISSN:1933-0219
Container-title:Mucosal Immunology
language:en
Short-container-title:Mucosal Immunol

Author:

Waldschmitt N,Berger E,Rath E,Sartor R B,Weigmann B,Heikenwalder M,Gerhard M,Janssen K-P,Haller D

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/mi201434.pdf

Reference50 articles.

1. McGovern, D.P. et al. Genome-wide association identifies multiple ulcerative colitis susceptibility loci. Nat. Genet. 42, 332–337 (2010).

2. Imielinski, M. et al. Common variants at five new loci associated with early-onset inflammatory bowel disease. Nat. Genet. 41, 1335–1340 (2009).

3. Maeda, S. et al. Nod2 mutation in Crohn's disease potentiates NF-kappaB activity and IL-1beta processing. Science 307, 734–738 (2005).

4. Kontoyiannis, D. et al. Genetic dissection of the cellular pathways and signaling mechanisms in modeled tumor necrosis factor-induced Crohn's-like inflammatory bowel disease. J. Exp. Med. 196, 1563–1574 (2002).

5. Muise, A.M. et al. Polymorphisms in E-cadherin (CDH1) result in a mis-localised cytoplasmic protein that is associated with Crohn's disease. Gut 58, 1121–1127 (2009).

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