Bcr–Abl activates the AKT/FoxO3 signalling pathway to restrict transforming growth factor‐β‐mediated cytostatic signals
Author:
Affiliation:
1. INSERM U482 184 Rue du Faubourg St‐Antoine 75571 Paris France
2. INSERM U 542 14 Avenue PV Couturier 94807 Villejuif France
Publisher
EMBO
Subject
Genetics,Molecular Biology,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1038/sj.embor.7400501
Reference22 articles.
1. Evidence for a Role of MSK1 in Transforming Growth Factor-β-mediated Responses through p38α and Smad Signaling Pathways
2. FoxOs at the Crossroads of Cellular Metabolism, Differentiation, and Transformation
3. Evidence for a role of NF- B in the survival of hematopoietic cells mediated by interleukin 3 and the oncogenic TEL/platelet-derived growth factor receptor fusion protein
4. Akt Promotes Cell Survival by Phosphorylating and Inhibiting a Forkhead Transcription Factor
5. Akt interacts directly with Smad3 to regulate the sensitivity to TGF-β-induced apoptosis
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