Activation of c-Met in cancer cells mediates growth-promoting signals against oxidative stress through Nrf2-HO-1-Reference-Cited by-同舟云学术

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Activation of c-Met in cancer cells mediates growth-promoting signals against oxidative stress through Nrf2-HO-1

Published:2019-01-15 Issue:2 Volume:8 Page:
ISSN:2157-9024
Container-title:Oncogenesis
language:en
Short-container-title:Oncogenesis

Author:

Chakraborty Samik,Balan Murugabaskar,Flynn Evelyn,Zurakowski David,Choueiri Toni K.,Pal Soumitro

Funder

U.S. Department of Health & Human Services | NIH | National Cancer Institute

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Molecular Biology

Link

http://www.nature.com/articles/s41389-018-0116-9.pdf

Reference54 articles.

1. Bielecka, Z. F. et al. Hypoxic 3D in vitro culture models reveal distinct resistance processes to TKIs in renal cancer cells. Cell Biosci. 7, 71 (2017).

2. Gherardi, E., Birchmeier, W., Birchmeier, C. & Vande Woude, G. Targeting MET in cancer: rationale and progress. Nat. Rev. Cancer 12, 89–103 (2012).

3. Pecuchet, N., Fournier, L. S. & Oudard, S. New insights into the management of renal cell cancer. Oncology 84, 22–31 (2013).

4. Giubellino, A., Linehan, W. M. & Bottaro, D. P. Targeting the Met signaling pathway in renal cancer. Expert Rev. Anticancer Ther. 9, 785–93 (2009).

5. Balan, M. et al. Novel roles of c-Met in the survival of renal cancer cells through the regulation of HO-1 and PD-L1 expression. J. Biol. Chem. 290, 8110–20 (2015).

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