RecQL4-Aurora B kinase axis is essential for cellular proliferation, cell cycle progression, and mitotic integrity
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Molecular Biology
Link
http://www.nature.com/articles/s41389-018-0080-4.pdf
Reference45 articles.
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2. Mann, M. B. et al. Defective sister-chromatid cohesion, aneuploidy and cancer predisposition in a mouse model of type II Rothmund–Thomson syndrome. Hum. Mol. Genet. 14, 813–825 (2005).
3. Kitao, S. et al. Mutations in RECQL4 cause a subset of cases of Rothmund–Thomson syndrome. Nat. Genet. 22, 82–84 (1999).
4. Croteau, D. L., Singh, D. K., Hoh Ferrarelli, L., Lu, H. & Bohr, V. A. RECQL4 in genomic instability and aging. Trends Genet. 28, 624–631 (2012).
5. Croteau, D. L., Popuri, V., Opresko, P. L. & Bohr, V. A. Human RecQ helicases in DNA repair, recombination, and replication. Annu. Rev. Biochem. 83, 519–552 (2014).
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