XK-related protein 5 (XKR5) is a novel negative regulator of KIT/D816V-mediated transformation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Molecular Biology
Link
http://www.nature.com/articles/s41389-018-0057-3.pdf
Reference21 articles.
1. Lennartsson, J. & Rönnstrand, L. Stem cell factor receptor/c-Kit: from basic science to clinical implications. Physiol. Rev. 92, 1619–1649 (2012).
2. Kazi, J. U., Agarwal, S., Sun, J., Bracco, E. & Rönnstrand, L. Src-like-adaptor protein (SLAP) differentially regulates normal and oncogenic c-Kit signaling. J. Cell Sci. 127, 653–662 (2014).
3. Piao, X., Paulson, R., van der Geer, P., Pawson, T. & Bernstein, A. Oncogenic mutation in the Kit receptor tyrosine kinase alters substrate specificity and induces degradation of the protein tyrosine phosphatase SHP-1. Proc. Natl Acad. Sci. USA 93, 14665–14669 (1996).
4. Sun, J. et al. The PI3-kinase isoform p110delta is essential for cell transformation induced by the D816V mutant of c-Kit in a lipid-kinase-independent manner. Oncogene 33, 5360–5369 (2014).
5. Sun, J., Pedersen, M. & Rönnstrand, L. The D816V mutation of c-Kit circumvents a requirement for Src family kinases in c-Kit signal transduction. J. Biol. Chem. 284, 11039–11047 (2009).
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2. Loss of PI3 kinase association improves the sensitivity of secondary mutation of KIT to Imatinib;Cell & Bioscience;2020-02-12
3. Phosphorylation-mediated activation of mouse Xkr8 scramblase for phosphatidylserine exposure;Proceedings of the National Academy of Sciences;2019-02-04
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