Epigenomic reprogramming during pancreatic cancer progression links anabolic glucose metabolism to distant metastasis-Reference-Cited by-同舟云学术

Epigenomic reprogramming during pancreatic cancer progression links anabolic glucose metabolism to distant metastasis

Published:2017-01-16 Issue:3 Volume:49 Page:367-376
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

McDonald Oliver G,Li Xin,Saunders Tyler,Tryggvadottir Rakel,Mentch Samantha J,Warmoes Marc O,Word Anna E,Carrer Alessandro,Salz Tal H,Natsume Sonoko,Stauffer Kimberly M,Makohon-Moore Alvin,Zhong Yi,Wu Hao,Wellen Kathryn E^ORCID,Locasale Jason W^ORCID,Iacobuzio-Donahue Christine A^ORCID,Feinberg Andrew P

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng.3753.pdf

Reference48 articles.

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3. Iacobuzio-Donahue, C.A. et al. DPC4 gene status of the primary carcinoma correlates with patterns of failure in patients with pancreatic cancer. J. Clin. Oncol. 27, 1806–1813 (2009).

4. Rahib, L. et al. Projecting cancer incidence and deaths to 2030: the unexpected burden of thyroid, liver, and pancreas cancers in the United States. Cancer Res. 74, 2913–2921 (2014).

5. Wen, B., Wu, H., Shinkai, Y., Irizarry, R.A. & Feinberg, A.P. Large histone H3 lysine 9 dimethylated chromatin blocks distinguish differentiated from embryonic stem cells. Nat. Genet. 41, 246–250 (2009).

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