Hypertrophic preconditioning attenuates post-myocardial infarction injury through deacetylation of isocitrate dehydrogenase 2-Reference-Cited by-同舟云学术

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Hypertrophic preconditioning attenuates post-myocardial infarction injury through deacetylation of isocitrate dehydrogenase 2

Published:2021-06-23 Issue:12 Volume:42 Page:2004-2015
ISSN:1671-4083
Container-title:Acta Pharmacologica Sinica
language:en
Short-container-title:Acta Pharmacol Sin

Author:

Ma Lei-lei,Kong Fei-juan,Ma Yuan-ji,Guo Jun-jie,Wang Shi-jun,Dong Zheng,Sun Ai-jun,Zou Yun-zeng,Ge Jun-bo

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology,General Medicine

Link

https://www.nature.com/articles/s41401-021-00699-0.pdf

Reference33 articles.

1. Hill JA, Olson EN. Cardiac plasticity. N Engl J Med. 2008;358:1370–80.

2. Cung TT, Morel O, Cayla G, Rioufol G, Garcia-Dorado D, Angoulvant D, et al. Cyclosporine before PCI in patients with acute myocardial infarction. N Engl J Med. 2015;373:1021–31.

3. Cadenas S. ROS and redox signaling in myocardial ischemia-reperfusion injury and cardioprotection. Free Radic Biol Med. 2018;117:76–89.

4. Pei H, Song X, Peng C, Tan Y, Li Y, Li X, et al. TNF-alpha inhibitor protects against myocardial ischemia/reperfusion injury via Notch1-mediated suppression of oxidative/nitrative stress. Free Radic Biol Med. 2015;82:114–21.

5. Ge H, Zhao M, Lee S, Xu Z. Mitochondrial Src tyrosine kinase plays a role in the cardioprotective effect of ischemic preconditioning by modulating complex I activity and mitochondrial ROS generation. Free Radic Res. 2015;49:1210–7.

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