Downregulated calmodulin expression contributes to endothelial cell impairment in diabetes-Reference-Cited by-同舟云学术

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Downregulated calmodulin expression contributes to endothelial cell impairment in diabetes

Published:2023-07-19 Issue: Volume: Page:
ISSN:1671-4083
Container-title:Acta Pharmacologica Sinica
language:en
Short-container-title:Acta Pharmacol Sin

Author:

Liu Tian-tian,Xu Huan-huan,Liu Ze-juan,Zhang He-ping,Zhou Hai-tao,Zhu Zhi-xiang,Wang Zhi-qiang,Xue Jing-yi,Li Qiang,Ma Yi,You Hong-jie,Luo Da-li

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology,General Medicine

Link

https://www.nature.com/articles/s41401-023-01127-1.pdf

Reference51 articles.

1. Sun H, Saeedi P, Karuranga S, Pinkepank M, Ogurtsova K, Duncan B, et al. IDF diabetes Atlas: global, regional and country-level diabetes prevalence estimates for 2021 and projections for 2045. Diabetes Res Clin Pr. 2021;183:109119.

2. Skyler J, Bakris G, Bonifacio E, Darsow T, Eckel R, Groop L, et al. Differentiation of diabetes by pathophysiology, natural history, and prognosis. Diabetes. 2017;66:241–55.

3. Giacco F, Brownlee M. Oxidative stress and diabetic complications. Circ Res. 2010;107:1058–70.

4. Su J, Lucchesi PA, Gonzalez-Villalobos RA, Palen DI, Rezk BM, Suzuki Y, et al. Role of advanced glycation end products with oxidative stress in resistance artery dysfunction in type 2 diabetic mice. Arterioscler Thromb Vasc Biol. 2008;28:1432–8.

5. Förstermann U, Sessa W. Nitric oxide synthases: regulation and function. Eur Heart J. 2012;33:829–37.

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2. Tibial cortex transverse transport regulates Orai1/STIM1-mediated NO release and improve the migration and proliferation of vessels via increasing osteopontin expression;Journal of Orthopaedic Translation;2024-03

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