Chromosome stability, in the absence of apoptosis, is critical for suppression of tumorigenesis in Trp53 mutant mice-Reference-Cited by-同舟云学术

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Chromosome stability, in the absence of apoptosis, is critical for suppression of tumorigenesis in Trp53 mutant mice

Published:2003-12-21 Issue:1 Volume:36 Page:63-68
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

Liu Geng,Parant John M,Lang Gene,Chau Patty,Chavez-Reyes Arturo,El-Naggar Adel K,Multani Asha,Chang Sandy,Lozano Guillermina

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng1282.pdf

Reference30 articles.

1. Ko, L.J. & Prives, C. p53: puzzle and paradigm. Genes Dev. 10, 1054–1072 (1996).

2. Levine, A.J. p53, the cellular gatekeeper for growth and division. Cell 88, 323–331 (1997).

3. Vogelstein, B., Lane, D. & Levine, A.J. Surfing the p53 network. Nature 408, 307–310 (2000).

4. Rowan, S. et al. Specific loss of apoptotic but not cell-cycle arrest function in a human tumor derived p53 mutant. EMBO J. 15, 827–838 (1996).

5. Ludwig, R.L., Bates, S. & Vousden, K.H. Differential activation of target cellular promoters by p53 mutants with impaired apoptotic function. Mol. Cell. Biol. 16, 4952–4960 (1996).

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