Dynein disruption perturbs post-synaptic components and contributes to impaired MuSK clustering at the NMJ: implication in ALS
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep27804.pdf
Reference71 articles.
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3. DeChiara, T. M. et al. The receptor tyrosine kinase MuSK is required for neuromuscular junction formation in vivo. Cell 85, 501–512 (1996).
4. Denzer, A. J., Gesemann, M., Schumacher, B. & Ruegg, M. A. An amino-terminal extension is required for the secretion of chick agrin and its binding to extracellular matrix. J. Cell Biol. 131, 1547–1560 (1995).
5. Glass, D. J. et al. Agrin acts via a MuSK receptor complex. Cell 85, 513–523 (1996).
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