Pro-proliferative FoxM1 is a target of p53-mediated repression
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Genetics,Molecular Biology
Link
http://www.nature.com/articles/onc2009282.pdf
Reference77 articles.
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3. Bhonde MR, Hanski ML, Budczies J, Cao M, Gillissen B, Moorthy D et al. (2006). DNA damage-induced expression of p53 suppresses mitotic checkpoint kinase hMps1: the lack of this suppression in p53MUT cells contributes to apoptosis. J Biol Chem 281: 8675–8685.
4. Blanco-Bose WE, Murphy MJ, Ehninger A, Offner S, Dubey C, Huang W et al. (2008). C-Myc and its target FoxM1 are critical downstream effectors of constitutive androstane receptor (CAR) mediated direct liver hyperplasia. Hepatology 48: 1302–1311.
5. Bunz F, Dutriaux A, Lengauer C, Waldman T, Zhou S, Brown JP et al. (1998). Requirement for p53 and p21 to sustain G2 arrest after DNA damage. Science 282: 1497–1501.
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