Aryl hydrocarbon receptor inhibition downregulates the TGF-β/Smad pathway in human glioblastoma cells-Reference-Cited by-同舟云学术

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Aryl hydrocarbon receptor inhibition downregulates the TGF-β/Smad pathway in human glioblastoma cells

Published:2009-05-25 Issue:28 Volume:28 Page:2593-2605
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Gramatzki D,Pantazis G,Schittenhelm J,Tabatabai G,Köhle C,Wick W,Schwarz M,Weller M,Tritschler I

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2009104.pdf

Reference47 articles.

1. Andersson P, McGuire J, Rubio C, Gradin K, Whitelaw ML, Pettersson S et al. (2002). A constitutively active dioxin/aryl hydrocarbon receptor induces stomach tumors. Proc Natl Acad Sci USA 99: 9990–9995.

2. Bruna A, Darken RS, Rojo F, Ocana A, Penuelas S, Arias A et al. (2007). High TGFbeta-Smad activity confers poor prognosis in glioma patients and promotes cell proliferation depending on the methylation of the PDGF-B gene. Cancer Cell 11: 147–160.

3. Carraro G, Albertin G, Forneris M, Nussdorfer GG . (2005). Similar sequence-free amplification of human glyceraldehyde-3-phosphate dehydrogenase for real time RT-PCR applications. Mol Cell Probes 19: 181–186.

4. Chang X, Fan Y, Karyala S, Schwemberger S, Tomlinson CR, Sartor MA et al. (2007). Ligand-independent regulation of transforming growth factor beta1 expression and cell cycle progression by the aryl hydrocarbon receptor. Mol Cell Biol 27: 6127–6139.

5. Corchero J, Martin-Partido G, Dallas SL, Fernandez-Salguero PM . (2004). Liver portal fibrosis in dioxin receptor-null mice that overexpress the latent transforming growth factor-beta-binding protein-1. Int J Exp Pathol 85: 295–302.

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