Phenotype-assisted transcriptome analysis identifies FOXM1 downstream from Ras–MKK3–p38 to regulate in vitro cellular invasion-Reference-Cited by-同舟云学术

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Phenotype-assisted transcriptome analysis identifies FOXM1 downstream from Ras–MKK3–p38 to regulate in vitro cellular invasion

Published:2009-12-21 Issue:10 Volume:29 Page:1519-1530
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Behren A,Mühlen S,Acuna Sanhueza G A,Schwager C,Plinkert P K,Huber P E,Abdollahi A,Simon C

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2009436.pdf

Reference39 articles.

1. Adam AP, George A, Schewe D, Bragado P, Iglesias BV, Ranganathan AC et al (2009). Computational identification of a p38SAPK-regulated transcription factor network required for tumor cell quiescence. Cancer Res 69: 5664–5672.

2. Behren A, Binder K, Vucelic G, Herberhold S, Hirt B, Loewenheim H et al (2005). The p38 SAPK pathway is required for Ha-ras induced in vitro invasion of NIH3T3 cells. Exp Cell Res 303: 321–330.

3. Bradley MO, Kraynak AR, Storer RD, Gibbs JB . (1986). Experimental metastasis in nude mice of NIH 3T3 cells containing various ras genes. Proc Natl Acad Sci USA 83: 5277–5281.

4. Demuth T, Reavie LB, Rennert JL, Nakada M, Nakada S, Hoelzinger DB et al (2007). MAP-ing glioma invasion: mitogen-activated protein kinase kinase 3 and p38 drive glioma invasion and progression and predict patient survival. Mol Cancer Ther 6: 1212–1222.

5. Deng Q, Liao R, Wu BL, Sun P . (2004). High intensity ras signaling induces premature senescence by activating p38 pathway in primary human fibroblasts. J Biol Chem 279: 1050–1059.

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