Histone deacetylase inhibitors induce thyroid cancer-specific apoptosis through proteasome-dependent inhibition of TRAIL degradation-Reference-Cited by-同舟云学术

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Histone deacetylase inhibitors induce thyroid cancer-specific apoptosis through proteasome-dependent inhibition of TRAIL degradation

Published:2009-10-05 Issue:1 Volume:29 Page:105-116
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Borbone E,Berlingieri M T,De Bellis F,Nebbioso A,Chiappetta G,Mai A,Altucci L,Fusco A

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2009306.pdf

Reference50 articles.

1. Altucci L, Clarke N, Nebbioso A, Scognamiglio A, Gronemeyer H . (2005). Acute myeloid leukemia: therapeutic impact of epigenetic drugs. Int J Biochem Cell Biol 37: 1752–1762.

2. Altucci L, Rossin A, Raffelsberger W, Reitmair A, Chomienne C, Gronemeyer H . (2001). Retinoic acid-induced apoptosis in leukemia cells is mediated by paracrine action of tumor-selective death ligand TRAIL. Nat Med 7: 680–686.

3. Altucci L, Stunnenberg HG . (2009). Time for epigenetics. Int J Biochem Cell Biol 41: 2–3.

4. Ashkenazi A . (2002). Targeting death and decoy receptors of the tumour-necrosis factor superfamily. Nat Rev Cancer 2: 420–430.

5. Blumenschein Jr GR., Kies MS, Papadimitrakopoulou VA, Lu C, Kumar AJ, Ricker JL et al (2008). Phase II trial of the histone deacetylase inhibitor vorinostat (Zolinza, suberoylanilide hydroxamic acid, SAHA) in patients with recurrent and/or metastatic head and neck cancer. Invest New Drugs 26: 81–87.

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