Three Epstein–Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints-Reference-Cited by-同舟云学术

Three Epstein–Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints

Published:2009-08-31 Issue:45 Volume:28 Page:3997-4008
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Gruhne B,Sompallae R,Masucci M G

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2009258.pdf

Reference54 articles.

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4. Bose S, Yap LF, Fung M, Starzcynski J, Saleh A, Morgan S et al. (2009). The ATM tumour suppressor gene is down-regulated in EBV-associated nasopharyngeal carcinoma. J Pathol 217: 345–352.

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