Functional interaction of HTLV-1 Tax protein with the POZ domain of the transcriptional repressor BCL6
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Genetics,Molecular Biology
Link
http://www.nature.com/articles/onc2009230.pdf
Reference64 articles.
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2. Albagli O, Lantoine D, Quief S, Quignon F, Englert C, Kerckaert JP et al. (1999). Overexpressed BCL6 (LAZ3) oncoprotein triggers apoptosis, delays S phase progression and associates with replication foci. Oncogene 18: 5063–5075.
3. Albagli O, Lindon C, Lantoine D, Quief S, Puvion E, Pinset C et al. (2000). DNA replication progresses on the periphery of nuclear aggregates formed by the BCL6 transcription factor. Mol Cell Biol 20: 8560–8570.
4. Alizadeh AA, Eisen MB, Davis RE, Ma C, Lossos IS, Rosenwald A et al. (2000). Distinct types of diffuse large B-cell lymphoma identified by gene expression profiling. Nature 403: 503–511.
5. Baron BW, Anastasi J, Thirman MJ, Furukawa Y, Fears S, Kim DC et al. (2002). The human programmed cell death-2 (PDCD2) gene is a target of BCL6 repression: implications for a role of BCL6 in the down-regulation of apoptosis. Proc Natl Acad Sci USA 99: 2860–2865.
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1. Visualizing spatiotemporal dynamics of apoptosis after G1 arrest by human T cell leukemia virus type 1 Tax and insights into gene expression changes using microarray-based gene expression analysis;BMC Genomics;2012-06-22
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