Hematopoietic stem cell ageing is uncoupled from p16INK4A-mediated senescence
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Genetics,Molecular Biology
Link
http://www.nature.com/articles/onc200994.pdf
Reference26 articles.
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2. Bracken AP, Kleine-Kohlbrecher D, Dietrich N, Pasini D, Gargiulo G, Beekman C et al. (2007). The Polycomb group proteins bind throughout the INK4A-ARF locus and are disassociated in senescent cells. Genes Dev 21: 525–530.
3. Bryder D, Rossi DJ, Weissman IL . (2006). Hematopoietic stem cells: the paradigmatic tissue-specific stem cell. Am J Pathol 169: 338–346.
4. Jacobs JJ, Kieboom K, Marino S, DePinho RA, van Lohuizen M . (1999). The oncogene and Polycomb-group gene bmi-1 regulates cell proliferation and senescence through the ink4a locus. Nature 397: 164–168.
5. Janzen V, Forkert R, Fleming HE, Saito Y, Waring MT, Dombkowski DM et al. (2006). Stem-cell ageing modified by the cyclin-dependent kinase inhibitor p16INK4a. Nature 443: 421–426.
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