Cardiac hypertrophy is inhibited by antagonism of ADAM12 processing of HB-EGF: Metalloproteinase inhibitors as a new therapy-Reference-Cited by-同舟云学术

Cardiac hypertrophy is inhibited by antagonism of ADAM12 processing of HB-EGF: Metalloproteinase inhibitors as a new therapy

Published:2002-01 Issue:1 Volume:8 Page:35-40
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Asakura Masanori,Kitakaze Masafumi,Takashima Seiji,Liao Yulin,Ishikura Fuminobu,Yoshinaka Tsuyoshi,Ohmoto Hiroshi,Node Koichi,Yoshino Kohichiro,Ishiguro Hiroshi,Asanuma Hiroshi,Sanada Shoji,Matsumura Yasushi,Takeda Hiroshi,Beppu Shintaro,Tada Michihiko,Hori Masatsugu,Higashiyama Shigeki

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm0102-35.pdf

Reference22 articles.

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2. Levy, D., Garrison, R.J., Savage, D.D., Kannel, W.B. & Castelli, W.P. Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study. N. Engl. J. Med 322, 1561–1566 (1990).

3. Simpson, P., McGrath, A. & Savion, S. Myocyte hypertrophy in neonatal rat heart cultures and its regulation by serum and by catecholamines. Circ. Res. 51, 787–801 (1982).

4. Ito, H. et al. Endothelin-1 induces hypertrophy with enhanced expression of muscle-specific genes in cultured neonatal rat cardiomyocytes. Circ. Res. 69, 209–215 (1991).

5. Sadoshima, J., Xu, Y., Slayter, H.S. & Izumo, S. Autocrine release of angiotensin II mediates stretch-induced hypertrophy of cardiac myocytes in vitro. Cell 75, 977–984 (1993).

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