SPHK1 deficiency protects mice from acetaminophen-induced ER stress and mitochondrial permeability transition-Reference-Cited by-同舟云学术

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SPHK1 deficiency protects mice from acetaminophen-induced ER stress and mitochondrial permeability transition

Published:2019-12-11 Issue:6 Volume:27 Page:1924-1937
ISSN:1350-9047
Container-title:Cell Death & Differentiation
language:en
Short-container-title:Cell Death Differ

Author:

Li Longjun,Wang Haitao,Zhang Jun,Sha Yeqin,Wu Fengjiao,Wen Shuang,He Lianping,Sheng Liang,You Qiang,Shi Meiqing,Liu Lixin,Zhou Hong

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/s41418-019-0471-x.pdf

Reference41 articles.

1. Ostapowicz G, Fontana RJ, Schiodt FV, Larson A, Davern TJ, Han SH, et al. Results of a prospective study of acute liver failure at 17 tertiary care centers in the United States. Ann Intern Med. 2002;137:947–54.

2. Schilling A, Corey R, Leonard M, Eghtesad B. Acetaminophen: old drug, new warnings. Clevel Clin J Med. 2010;77:19–27.

3. Lee WM. Acetaminophen toxicity: changing perceptions on a social/medical issue. Hepatology. 2007;46:966–70.

4. Dahlin DC, Miwa GT, Lu AY, Nelson SD. N-acetyl-p-benzoquinone imine: a cytochrome P-450-mediated oxidation product of acetaminophen. Proc Natl Acad Sci USA. 1984;81:1327–31.

5. Coles B, Wilson I, Wardman P, Hinson JA, Nelson SD, Ketterer B. The spontaneous and enzymatic reaction of N-acetyl-p-benzoquinonimine with glutathione: a stopped-flow kinetic study. Arch Biochem Biophys. 1988;264:253–60.

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