In the absence of apoptosis, myeloid cells arrest when deprived of growth factor, but remain viable by consuming extracellular glucose-Reference-Cited by-同舟云学术

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In the absence of apoptosis, myeloid cells arrest when deprived of growth factor, but remain viable by consuming extracellular glucose

Published:2019-02-15 Issue:10 Volume:26 Page:2074-2085
ISSN:1350-9047
Container-title:Cell Death & Differentiation
language:en
Short-container-title:Cell Death Differ

Author:

Dong Li,Reljic Boris,Cheung Jen G.,Ng Elizabeth S.,Lindqvist Lisa M.^ORCID,Elefanty Andrew G.,Vaux David L.^ORCID,Tran Hoanh^ORCID

Funder

Department of Health | National Health and Medical Research Council

Leukemia and Lymphoma Society

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/s41418-019-0275-z.pdf

Reference24 articles.

1. Perkins AC, Cory S. Conditional immortalization of mouse myelomonocytic, megakaryocytic and mast cell progenitors by the Hox-2.4 homeobox gene. EMBO J. 1993;12:3835–46.

2. Ekert PG, Read SH, Silke J, Marsden VS, Kaufmann H, Hawkins CJ, et al. Apaf-1 and caspase-9 accelerate apoptosis, but do not determine whether factor-deprived or drug-treated cells die. J Cell Biol. 2004;165:835–42.

3. Vaux DL, Cory S, Adams JM. Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cells. Nature. 1988;335:440–2.

4. Lum JJ, Bauer DE, Kong M, Harris MH, Li C, Lindsten T, et al. Growth factor regulation of autophagy and cell survival in the absence of apoptosis. Cell. 2005;120:237–48.

5. Mizushima N, Komatsu M. Autophagy: renovation of cells and tissues. Cell. 2011;147:728–41.https://doi.org/710.1016/j.cell.2011.1010.1026.

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