The brace helices of MLKL mediate interdomain communication and oligomerisation to regulate cell death by necroptosis-Reference-Cited by-同舟云学术

The brace helices of MLKL mediate interdomain communication and oligomerisation to regulate cell death by necroptosis

Published:2018-02-14 Issue:9 Volume:25 Page:1567-1580
ISSN:1350-9047
Container-title:Cell Death & Differentiation
language:en
Short-container-title:Cell Death Differ

Author:

Davies Katherine A.,Tanzer Maria C.,Griffin Michael D. W.,Mok Yee Foong,Young Samuel N.,Qin Rui,Petrie Emma J.,Czabotar Peter E.,Silke John^ORCID,Murphy James M.

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/s41418-018-0061-3.pdf

Reference44 articles.

1. Murphy JM, Czabotar PE, Hildebrand JM, Lucet IS, Zhang JG, Alvarez-Diaz S, et al. The pseudokinase MLKL mediates necroptosis via a molecular switch mechanism. Immunity. 2013;39:443–53.

2. Sun L, Wang H, Wang Z, He S, Chen S, Liao D, et al. Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase. Cell. 2012;148:213–27.

3. Wu J, Huang Z, Ren J, Zhang Z, He P, Li Y, et al. Mlkl knockout mice demonstrate the indispensable role of Mlkl in necroptosis. Cell Res. 2013;23:994–1006.

4. Zhao J, Jitkaew S, Cai Z, Choksi S, Li Q, Luo J, et al. Mixed lineage kinase domain-like is a key receptor interacting protein 3 downstream component of TNF-induced necrosis. Proc Natl Acad Sci USA. 2012;109:5322–7.

5. Silke J, Rickard JA, Gerlic M. The diverse role of RIP kinases in necroptosis and inflammation. Nat Immunol. 2015;16:689–97.

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