Salt wasting in distal renal tubular acidosis—new look, old problem
Author:
Publisher
Springer Science and Business Media LLC
Subject
Nephrology
Link
http://www.nature.com/articles/nrneph.2013.235.pdf
Reference9 articles.
1. Sartorius, O. W., Roemmelt, J. C. & Pitts, R. F. The renal regulation of acid-base balance in man; the nature of the renal compensations in ammonium chloride acidosis. J. Clin. Invest. 28, 423–439 (1949).
2. Sebastian, A., McSherry, E. & Morris, R. C. Jr. Renal potassium wasting in renal tubular acidosis (RTA): its occurrence in types 1 and 2 RTA despite sustained correction of systemic acidosis. J. Clin. Invest. 50, 667–678 (1971).
3. Gueutin, V. et al. Renal β-intercalated cells maintain body fluid and electrolyte balance. J. Clin. Invest. 123, 4219–4231 (2013).
4. Leviel, F. et al. The Na+-dependent chloride-bicarbonate exchanger SLC4A8 mediates an electroneutral Na+ reabsorption process in the renal cortical collecting ducts of mice. J. Clin. Invest. 120, 1627–1635 (2010).
5. Ismail-Beigi, F. & Edelman, I. S. The mechanism of the calorigenic action of thyroid hormone. Stimulation of Na+ + K+-activated adenosinetriphosphatase activity. J. Gen. Physiol. 57, 710–722 (1971).
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