CD36 in chronic kidney disease: novel insights and therapeutic opportunities
Author:
Publisher
Springer Science and Business Media LLC
Subject
Nephrology
Link
http://www.nature.com/articles/nrneph.2017.126.pdf
Reference199 articles.
1. Nicholson, A. C., Febbraio, M., Han, J., Silverstein, R. L. & Hajjar, D. P. CD36 in atherosclerosis. The role of a class B macrophage scavenger receptor. Ann. N. Y. Acad. Sci. 902, 128–133 (2000).
2. Miquilena-Colina, M. E. et al. Hepatic fatty acid translocase CD36 upregulation is associated with insulin resistance, hyperinsulinaemia and increased steatosis in non-alcoholic steatohepatitis and chronic hepatitis C. Gut 60, 1394–1402 (2011).
3. Handberg, A. et al. Soluble CD36 (sCD36) clusters with markers of insulin resistance, and high sCD36 is associated with increased type 2 diabetes risk. J. Clin. Endocrinol. Metab. 95, 1939–1946 (2010).
4. Pascual, G. et al. Targeting metastasis-initiating cells through the fatty acid receptor CD36. Nature 541, 41–45 (2017).
5. Jiang, Y. et al. Dyslipidemia in human kidney transplant recipients receiving cyclosporine and tacrolimus is associated with different expression of CD36 on peripheral blood monocytes. Transplant. Proc. 43, 1612–1615 (2011).
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