Immunomodulatory drugs disrupt the cereblon–CD147–MCT1 axis to exert antitumor activity and teratogenicity-Reference-Cited by-同舟云学术

Immunomodulatory drugs disrupt the cereblon–CD147–MCT1 axis to exert antitumor activity and teratogenicity

Published:2016-06-13 Issue:7 Volume:22 Page:735-743
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Eichner Ruth,Heider Michael,Fernández-Sáiz Vanesa,van Bebber Frauke,Garz Anne-Kathrin,Lemeer Simone,Rudelius Martina,Targosz Bianca-Sabrina,Jacobs Laura,Knorn Anna-Maria,Slawska Jolanta,Platzbecker Uwe,Germing Ulrich,Langer Christian,Knop Stefan,Einsele Herrmann,Peschel Christian,Haass Christian,Keller Ulrich,Schmid Bettina,Götze Katharina S,Kuster Bernhard,Bassermann Florian

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm.4128.pdf

Reference53 articles.

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3. Bartlett, J.B., Dredge, K. & Dalgleish, A.G. The evolution of thalidomide and its IMiD derivatives as anticancer agents. Nat. Rev. Cancer 4, 314–322 (2004).

4. Ito, T. et al. Identification of a primary target of thalidomide teratogenicity. Science 327, 1345–1350 (2010).

5. Zhu, Y.X. et al. Cereblon expression is required for the anti-myeloma activity of lenalidomide and pomalidomide. Blood 118, 4771–4779 (2011).

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