ATM specifically mediates repair of double-strand breaks with blocked DNA ends-Reference-Cited by-同舟云学术

ATM specifically mediates repair of double-strand breaks with blocked DNA ends

Published:2014-02-27 Issue:1 Volume:5 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Álvarez-Quilón Alejandro,Serrano-Benítez Almudena,Ariel Lieberman Jenna,Quintero Cristina,Sánchez-Gutiérrez Daniel,Escudero Luis M.,Cortés-Ledesma Felipe

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms4347.pdf

Reference44 articles.

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2. Lavin, M. F. Ataxia-telangiectasia: from a rare disorder to a paradigm for cell signalling and cancer. Nat. Rev. Mol. Cell. Biol. 9, 759–769 (2008).

3. McKinnon, P. J. ATM and the molecular pathogenesis of ataxia telangiectasia. Ann. Rev. Pathol. 7, 303–321 (2012).

4. Shiloh, Y. & Ziv, Y. The ATM protein kinase: regulating the cellular response to genotoxic stress, and more. Nat. Rev. Mol. Cell. Biol. 14, 197–210 (2013).

5. Reynolds, J. J. & Stewart, G. S. A nervous predisposition to unrepaired DNA double strand breaks. DNA Repair (Amst) 12, 588–599 (2013).

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