Loss of NDRG2 expression activates PI3K-AKT signalling via PTEN phosphorylation in ATLL and other cancers-Reference-Cited by-同舟云学术

Loss of NDRG2 expression activates PI3K-AKT signalling via PTEN phosphorylation in ATLL and other cancers

Published:2014-02-26 Issue:1 Volume:5 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Nakahata Shingo,Ichikawa Tomonaga,Maneesaay Phudit,Saito Yusuke,Nagai Kentaro,Tamura Tomohiro,Manachai Nawin,Yamakawa Norio,Hamasaki Makoto,Kitabayashi Issay,Arai Yasuhito,Kanai Yae,Taki Tomohiko,Abe Takaya,Kiyonari Hiroshi,Shimoda Kazuya,Ohshima Koichi,Horii Akira,Shima Hiroshi,Taniwaki Masafumi,Yamaguchi Ryoji,Morishita Kazuhiro

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms4393.pdf

Reference58 articles.

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3. Yasunaga, J. & Matsuoka, M. Leukaemogenic mechanism of human T-cell leukaemia virus type I. Rev. Med. Virol. 17, 301–311 (2007).

4. Fukuda, R. et al. Alteration of phosphatidylinositol 3-kinase cascade in the multilobulated nuclear formation of adult T cell leukemia/lymphoma (ATLL). Proc. Natl Acad. Sci. USA 102, 15213–15218 (2005).

5. Ikezoe, T. et al. Longitudinal inhibition of PI3K/Akt/mTOR signaling by LY294002 and rapamycin induces growth arrest of adult T-cell leukemia cells. Leuk. Res. 31, 673–682 (2007).

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