The rLrp of Mycobacterium tuberculosis inhibits proinflammatory cytokine production and downregulates APC function in mouse macrophages via a TLR2-mediated PI3K/Akt pathway activation-dependent mechanism-Reference-Cited by-同舟云学术

The rLrp of Mycobacterium tuberculosis inhibits proinflammatory cytokine production and downregulates APC function in mouse macrophages via a TLR2-mediated PI3K/Akt pathway activation-dependent mechanism

Published:2015-07-13 Issue:6 Volume:13 Page:729-745
ISSN:1672-7681
Container-title:Cellular & Molecular Immunology
language:en
Short-container-title:Cell Mol Immunol

Author:

Liu Yuan,Li Jia-Yun,Chen Su-Ting,Huang Hai-Rong,Cai Hong

Publisher

Springer Science and Business Media LLC

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/cmi201558.pdf

Reference91 articles.

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3. Hingley-Wilson SM, Sly LM, Reiner NE, McMaster WR . The immunobiology of the mycobacterial infected macrophage. Mod Aspects Immunobiol 2000; 1: 96–101.

4. Fenton MJ, Vermeulen MW . Immunopathology of tuberculosis: roles of macrophages and monocytes. Infect Immun 1996; 64: 683–690.

5. Armstrong JA, Hart PD . Phagosome-lysosome interactions in cultured macrophages infected with virulent tubercle bacilli. Reversal of the usual nonfusion pattern and observations on bacterial survival. J Exp Med 1975; 142: 1–16.

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