HAX1 deletion impairs BCR internalization and leads to delayed BCR-mediated apoptosis-Reference-Cited by-同舟云学术

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HAX1 deletion impairs BCR internalization and leads to delayed BCR-mediated apoptosis

Published:2015-04-13 Issue: Volume: Page:
ISSN:1672-7681
Container-title:Cellular and Molecular Immunology
language:
Short-container-title:Cell Mol Immunol

Author:

Wolkerstorfer Susanne,Schwaiger Elisabeth,Rinnerthaler Mark,Karina Gratz Iris,Zoegg Thomas,Brandstetter Hans,Achatz-Straussberger Gertrude

Publisher

Springer Science and Business Media LLC

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference54 articles.

1. Restoration of surface IgM-mediated apoptosis in an anti-IgM-resistant variant of WEHI-231 lymphoma cells by HS1, a protein-tyrosine kinase substrate.

2. Antigen-receptor induced clonal expansion and deletion of lymphocytes are impaired in mice lacking HS1 protein, a substrate of the antigen-receptor-coupled tyrosine kinases.

3. Role of Tyrosine Phosphorylation of HS1 in B Cell Antigen Receptor-mediated Apoptosis

4. The polycystic kidney disease protein PKD2 interacts with Hax-1, a protein associated with the actin cytoskeleton

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Kostmann’s Disease and HCLS1-Associated Protein X-1 (HAX1);Journal of Clinical Immunology;2016-12-10

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