Loss of Desmoglein Binding Is Not Sufficient for Keratinocyte Dissociation in Pemphigus
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Dermatology,Molecular Biology,Biochemistry
Reference49 articles.
1. The molecular architecture of cadherins in native epidermal desmosomes;Al-Amoudi;Nature,2007
2. Pemphigus vulgaris-IgG causes a rapid depletion of desmoglein 3 (Dsg3) from the Triton X-100 soluble pools, leading to the formation of Dsg3-depleted desmosomes in a human squamous carcinoma cell line, DJM-1 cells;Aoyama;J Invest Dermatol,1999
3. Induction of p38MAPK and HSP27 phosphorylation in pemphigus patient skin;Berkowitz;J Invest Dermatol,2008
4. Desmosome signaling. Inhibition of p38MAPK prevents pemphigus vulgaris IgG-induced cytoskeleton reorganization;Berkowitz;J Biol Chem,2005
5. p38MAPK inhibition prevents disease in pemphigus vulgaris mice;Berkowitz;Proc Natl Acad Sci USA,2006
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