Modelling Fanconi anemia pathogenesis and therapeutics using integration-free patient-derived iPSCs-Reference-Cited by-同舟云学术

Modelling Fanconi anemia pathogenesis and therapeutics using integration-free patient-derived iPSCs

Published:2014-07-07 Issue:1 Volume:5 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Liu Guang-Hui,Suzuki Keiichiro,Li Mo,Qu Jing,Montserrat Nuria,Tarantino Carolina,Gu Ying,Yi Fei,Xu Xiuling,Zhang Weiqi,Ruiz Sergio,Plongthongkum Nongluk,Zhang Kun,Masuda Shigeo,Nivet Emmanuel,Tsunekawa Yuji,Soligalla Rupa Devi,Goebl April,Aizawa Emi,Kim Na Young,Kim Jessica,Dubova Ilir,Li Ying,Ren Ruotong,Benner Chris,del Sol Antonio,Bueren Juan,Trujillo Juan Pablo,Surralles Jordi,Cappelli Enrico,Dufour Carlo,Esteban Concepcion Rodriguez,Belmonte Juan Carlos Izpisua

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms5330.pdf

Reference70 articles.

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2. Zhang, Q. S. et al. Tempol protects against oxidative damage and delays epithelial tumor onset in Fanconi anemia mice. Cancer Res. 68, 1601–1608 (2008).

3. Bogliolo, M. et al. Mutations in ERCC4, encoding the DNA-repair endonuclease XPF, cause Fanconi anemia. Am. J. Hum. Genet. 92, 800–806 (2013).

4. Moldovan, G. L. & D'Andrea, A. D. How the fanconi anemia pathway guards the genome. Annu. Rev. Genet. 43, 223–249 (2009).

5. Ceccaldi, R. et al. Bone marrow failure in fanconi anemia is triggered by an exacerbated p53/p21 DNA damage response that impairs hematopoietic stem and progenitor cells. Cell Stem Cell 11, 36–49 (2012).

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