SKLB-677, an FLT3 and Wnt/β-catenin signaling inhibitor, displays potent activity in models of FLT3-driven AML-Reference-Cited by-同舟云学术

SKLB-677, an FLT3 and Wnt/β-catenin signaling inhibitor, displays potent activity in models of FLT3-driven AML

Published:2015-10-26 Issue:1 Volume:5 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Ma Shuang,Yang Ling-Ling,Niu Ting,Cheng Chuan,Zhong Lei,Zheng Ming-Wu,Xiong Yu,Li Lin-Li,Xiang Rong,Chen Li-Juan,Zhou Qiao,Wei Yu-Quan,Yang Sheng-Yong

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep15646.pdf

Reference43 articles.

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3. Loriaux, M. M. et al. High-throughput sequence analysis of the tyrosine kinome in acute myeloid leukemia. Blood 111, 4788–4796 (2008).

4. Gilliland, D. G. & Griffin, J. D. The roles of FLT3 in hematopoiesis and leukemia. Blood 100, 1532–1542 (2002).

5. Reindl, C. et al. Point mutations in the juxtamembrane domain of FLT3 define a new class of activating mutations in AML. Blood 107, 3700–3707 (2006).

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