Rebound HIV-1 in cerebrospinal fluid after antiviral therapy interruption is mainly clonally amplified R5 T cell-tropic virus-Reference-Cited by-同舟云学术

Rebound HIV-1 in cerebrospinal fluid after antiviral therapy interruption is mainly clonally amplified R5 T cell-tropic virus

Published:2023-01-30 Issue: Volume: Page:
ISSN:2058-5276
Container-title:Nature Microbiology
language:en
Short-container-title:Nat Microbiol

Author:

Kincer Laura P.,Joseph Sarah Beth,Gilleece Maria M.,Hauser Blake M.,Sizemore Sabrina,Zhou Shuntai,Di Germanio Clara,Zetterberg Henrik^ORCID,Fuchs Dietmar^ORCID,Deeks Steven G.^ORCID,Spudich Serena,Gisslen Magnus,Price Richard W.^ORCID,Swanstrom Ronald^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Microbiology (medical),Genetics,Applied Microbiology and Biotechnology,Immunology,Microbiology

Link

https://www.nature.com/articles/s41564-022-01306-6.pdf

Reference69 articles.

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2. Parrish, N. F. et al. Transmitted/founder and chronic subtype C HIV-1 use CD4 and CCR5 receptors with equal efficiency and are not inhibited by blocking the integrin α4β7. PLoS Pathog. 8, e1002686 (2012).

3. Joseph, S. B. & Swanstrom, R. The evolution of HIV-1 entry phenotypes as a guide to changing target cells. J. Leuk. Biol. 103, 421–431 (2018).

4. Colby, D. J. et al. Rapid HIV RNA rebound after antiretroviral treatment interruption in persons durably suppressed in Fiebig I acute HIV infection. Nat. Med. 24, 923–926 (2018).

5. Henrich, T. J. et al. HIV-1 persistence following extremely early initiation of antiretroviral therapy (ART) during acute HIV-1 infection: an observational study. PLoS Med. 14, e1002417 (2017).

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