Microbiota-accessible carbohydrates suppress Clostridium difficile infection in a murine model
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Microbiology (medical),Genetics,Applied Microbiology and Biotechnology,Immunology,Microbiology
Link
http://www.nature.com/articles/s41564-018-0150-6.pdf
Reference54 articles.
1. Lessa, F. C., Winston, L. G. & McDonald, L. C. Burden of Clostridium difficile infection in the United States. N. Engl. J. Med. 372, 2369–2370 (2015).
2. Hryckowian, A. J., Pruss, K. M. & Sonnenburg, J. L. The emerging metabolic view of Clostridium difficile pathogenesis. Curr. Opin. Microbiol. 35, 42–47 (2017).
3. Khanna, S. et al. The epidemiology of community-acquired Clostridium difficile infection: a population-based study. Am. J. Gastroenterol. 107, 89–95 (2012).
4. Ng, K. M. et al. Microbiota-liberated host sugars facilitate post-antibiotic expansion of enteric pathogens. Nature 502, 96–99 (2013).
5. Ferreyra, J. A. et al. Gut microbiota-produced succinate promotes C. difficile infection after antibiotic treatment or motility disturbance. Cell Host Microbe 16, 770–777 (2014).
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