Intranasal delivery of the cytoplasmic domain of CTLA-4 using a novel protein transduction domain prevents allergic inflammation-Reference-Cited by-同舟云学术

Intranasal delivery of the cytoplasmic domain of CTLA-4 using a novel protein transduction domain prevents allergic inflammation

Published:2006-04-09 Issue:5 Volume:12 Page:574-579
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Choi Je-Min,Ahn Mi-Hyun,Chae Wook-Jin,Jung Yung-Gook,Park Jae-Chul,Song Hyun-Mi,Kim Young-Eun,Shin Jung-Ah,Park Choon-Sik,Park Jung-Won,Park Tae-Kwann,Lee Jung-Hoon,Seo Byung-Fhy,Kim Kyun-Do,Kim Eun-Sung,Lee Dong-Ho,Lee Seung-Kyou,Lee Sang-Kyou

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm1385.pdf

Reference30 articles.

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2. Wills-Karp, M. Interleukin-13 in asthma pathogenesis. Immunol. Rev. 202, 175–190 (2004).

3. Wills-Karp, M. Immunologic basis of antigen-induced airway hyperresponsiveness. Annu. Rev. Immunol. 17, 255–281 (1999).

4. Nakajima, H. et al. CD4+ T-lymphocytes and interleukin-5 mediate antigen-induced eosinophil infiltration into the mouse trachea. Am. Rev. Respir. Dis. 146, 374–377 (1992).

5. Yi, L.A., Hajialiasgar, S. & Chuang, E. Tyrosine-mediated inhibitory signals contribute to CTLA-4 function in vivo. Int. Immunol. 16, 539–547 (2004).

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