Trans-synaptic shift in anion gradient in spinal lamina I neurons as a mechanism of neuropathic pain-Reference-Cited by-同舟云学术

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Trans-synaptic shift in anion gradient in spinal lamina I neurons as a mechanism of neuropathic pain

Published:2003-08 Issue:6951 Volume:424 Page:938-942
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Coull Jeffrey A. M.,Boudreau Dominic,Bachand Karine,Prescott Steven A.,Nault Francine,Sík Attila,Koninck Paul De,Koninck Yves De

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature01868.pdf

Reference30 articles.

1. Melzack, R. & Wall, P. D. Pain mechanisms: a new theory. Science 150, 971–979 (1965)

2. Woolf, C. J. & Salter, M. W. Neuronal plasticity: increasing the gain in pain. Science 288, 1765–1769 (2000)

3. Kontinen, V. K., Stanfa, L. C., Basu, A. & Dickenson, A. H. Electrophysiologic evidence for increased endogenous GABAergic but not glycinergic inhibitory tone in the rat spinal nerve ligation model of neuropathy. Anesthesiology 94, 333–339 (2001)

4. Moore, K. A. et al. Partial peripheral nerve injury promotes a selective loss of GABAergic inhibition in the superficial dorsal horn of the spinal cord. J. Neurosci. 22, 6724–6731 (2002)

5. Somers, D. L. & Clemente, F. R. Dorsal horn synaptosomal content of aspartate, glutamate, glycine and GABA are differentially altered following chronic constriction injury to the rat sciatic nerve. Neurosci. Lett. 323, 171–174 (2002)

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