Telomere dysfunction and Atm deficiency compromises organ homeostasis and accelerates ageing-Reference-Cited by-同舟云学术

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Telomere dysfunction and Atm deficiency compromises organ homeostasis and accelerates ageing

Published:2003-01-22 Issue:6923 Volume:421 Page:643-648
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Wong Kwok-Kin,Maser Richard S.,Bachoo Robert M.,Menon Jayant,Carrasco Daniel R.,Gu Yansong,Alt Frederick W.,DePinho Ronald A.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature01385.pdf

Reference30 articles.

1. Shiloh, Y. & Kastan, M. B. ATM: genome stability, neuronal development, and cancer cross paths. Adv. Cancer Res. 83, 209–254 (2001)

2. Lee, H. W. et al. Essential role of mouse telomerase in highly proliferative organs. Nature 392, 569–574 (1998)

3. Blasco, M. A. et al. Telomere shortening and tumour formation by mouse cells lacking telomerase RNA. Cell 91, 25–34 (1997)

4. Borghesani, P. R. et al. Abnormal development of Purkinje cells and lymphocytes in Atm mutant mice. Proc. Natl Acad. Sci. USA 97, 3336–3341 (2000)

5. Kastan, M. B. & Lim, D. S. The many substrates and functions of ATM. Nature Rev. Mol. Cell Biol. 1, 179–186 (2000)

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