PAQR3 depletion accelerates diabetic wound healing by promoting angiogenesis through inhibiting STUB1-mediated PPARγ degradation
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Pathology and Forensic Medicine
Link
https://www.nature.com/articles/s41374-022-00786-8.pdf
Reference36 articles.
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3. Fui L.W., Lok M.P.W., Govindasamy V., Yong T.K., Lek T.K., Das A.K. Understanding the multifaceted mechanisms of diabetic wound healing and therapeutic application of stem cells conditioned medium in the healing process. J. Tissue Eng. Regen. Med. 13, 2218–2233 (2019).
4. Uccioli L., Izzo V., Meloni M., Vainieri E., Ruotolo V., Giurato L. Non-healing foot ulcers in diabetic patients: general and local interfering conditions and management options with advanced wound dressings. J. Wound Care24, 35–42 (2015).
5. Vijayakumar V., Samal S.K., Mohanty S., Nayak S.K. Recent advancements in biopolymer and metal nanoparticle-based materials in diabetic wound healing management. Int. J. Biol. Macromol.122, 137–148 (2019).
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1. METTL14 derived from exosomes of M1 macrophages promotes high glucose-induced apoptosis, inflammation and oxidative stress in glomerular endothelial cells by mediating PAQR3 m6A modification;Clinical and Experimental Nephrology;2024-07-30
2. The Combination of Vascular Endothelial Growth Factor A (VEGF-A) and Fibroblast Growth Factor 1 (FGF1) Modified mRNA Improves Wound Healing in Diabetic Mice: An Ex Vivo and In Vivo Investigation;Cells;2024-02-27
3. Systematic pan-cancer analyses of the potential function of the Golgi scaffold protein PAQR3;Scientific Reports;2024-02-06
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