Increasing nitric oxide bioavailability fails to improve collateral vessel formation in humanized sickle cell mice
Author:
Funder
U.S. Department of Health & Human Services | NIH | National Heart, Lung, and Blood Institute
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Pathology and Forensic Medicine
Link
https://www.nature.com/articles/s41374-022-00780-0.pdf
Reference48 articles.
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2. Large-vessel occlusion in sickle cell disease: pathogenesis, clinical consequences, and therapeutic implications;Francis;Med. Hypotheses.,1991
3. Pulmonary complications of sickle cell disease;Miller;Am. J. Respir. Crit. Care. Med.,2012
4. Sickle cell disease and the kidney;Scheinman;Nat. Clin. Pract. Nephrol.,2009
5. Impaired collateral vessel formation in sickle cell disease;Okwan-Duodu;Arterioscler Thromb. Vasc. Biol.,2018
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