SnoN residue (1–366) attenuates hypertrophic scars through resistance to transforming growth factor-β1-induced degradation-Reference-Cited by-同舟云学术

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SnoN residue (1–366) attenuates hypertrophic scars through resistance to transforming growth factor-β1-induced degradation

Published:2019-08-13 Issue:12 Volume:99 Page:1861-1873
ISSN:0023-6837
Container-title:Laboratory Investigation
language:en
Short-container-title:Lab Invest

Author:

Sun Gui-fang,Li Hong-chang,Zhan Yue-ping,Zhang Xiao-fen,Pan Li-yun,Chen Ya-feng,Xu Ke,Feng Dian-xu

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Link

http://www.nature.com/articles/s41374-019-0302-1.pdf

Reference53 articles.

1. Gauglitz GG, Korting HC, Pavicic T, Ruzicka T, Jeschke MG. Hypertrophic scarring and keloids: pathomechanisms and current and emerging treatment strategies. Mol Med. 2011;17:113.

2. Zhu Z, Ding J, Shankowsky HA, Tredget EE. The molecular mechanism of hypertrophic scar. J Cell Commun Signal. 2013;7:239–52.

3. Li B, Wang JH-C. Fibroblasts and myofibroblasts in wound healing: force generation and measurement. J Tissue Viability. 2011;20:108–20.

4. Rönty MJ, Leivonen S-K, Hinz B, Rachlin A, Otey CA, Kähäri V-M, et al. Isoform-specific regulation of the actin-organizing protein palladin during TGF-β1-induced myofibroblast differentiation. J Investi Dermatol. 2006;126:2387–96.

5. Leask A, Abraham DJ. TGF-β signaling and the fibrotic response. FASEB J. 2004;18:816–27.

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3. USP15 Enhances the Proliferation, Migration, and Collagen Deposition of Hypertrophic Scar–Derived Fibroblasts by Deubiquitinating TGF-βR1 In Vitro;Plastic & Reconstructive Surgery;2021-09-20

4. Panax Notoginseng Saponins suppresses TRPM7 via the PI3K/AKT pathway to inhibit hypertrophic scar formation in vitro;Burns;2021-06

5. The Ubiquitin Proteasome System and Skin Fibrosis;Molecular Diagnosis & Therapy;2021-01

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