The adaptor Act1 is required for interleukin 17–dependent signaling associated with autoimmune and inflammatory disease-Reference-Cited by-同舟云学术

The adaptor Act1 is required for interleukin 17–dependent signaling associated with autoimmune and inflammatory disease

Published:2007-02-04 Issue:3 Volume:8 Page:247-256
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Qian Youcun,Liu Caini,Hartupee Justin,Altuntas Cengiz Zubeyir,Gulen Muhammet Fatih,Jane-wit Daniel,Xiao Jianhua,Lu Yi,Giltiay Natalia,Liu Jinbo,Kordula Tomasz,Zhang Qi-Wei,Vallance Bruce,Swaidani Shadi,Aronica Mark,Tuohy Vincent K,Hamilton Thomas,Li Xiaoxia

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/ni1439.pdf

Reference55 articles.

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2. Veldhoen, M., Hocking, R.J., Atkins, C.J., Locksley, R.M. & Stockinger, B. TGFβ in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. Immunity 24, 179–189 (2006).

3. Mangan, P.R. et al. Transforming growth factor-β induces development of the TH17 lineage. Nature 441, 231–234 (2006).

4. Harrington, L.E. et al. Interleukin 17–producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages. Nat. Immunol. 6, 1123–1132 (2005).

5. Park, H. et al. A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17. Nat. Immunol. 6, 1133–1141 (2005).

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