Distinct classes of chromosomal rearrangements create oncogenic ETS gene fusions in prostate cancer-Reference-Cited by-同舟云学术

Distinct classes of chromosomal rearrangements create oncogenic ETS gene fusions in prostate cancer

Published:2007-08 Issue:7153 Volume:448 Page:595-599
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Tomlins Scott A.,Laxman Bharathi,Dhanasekaran Saravana M.,Helgeson Beth E.,Cao Xuhong,Morris David S.,Menon Anjana,Jing Xiaojun,Cao Qi,Han Bo,Yu Jindan,Wang Lei,Montie James E.,Rubin Mark A.,Pienta Kenneth J.,Roulston Diane,Shah Rajal B.,Varambally Sooryanarayana,Mehra Rohit,Chinnaiyan Arul M.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature06024.pdf

Reference34 articles.

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2. Tomlins, S. A. et al. TMPRSS2:ETV4 gene fusions define a third molecular subtype of prostate cancer. Cancer Res. 66, 3396–3400 (2006)

3. Cerveira, N. et al. TMPRSS2–ERG gene fusion causing ERG overexpression precedes chromosome copy number changes in prostate carcinomas and paired HGPIN lesions. Neoplasia 8, 826–832 (2006)

4. Glinsky, G. V., Glinskii, A. B., Stephenson, A. J., Hoffman, R. M. & Gerald, W. L. Gene expression profiling predicts clinical outcome of prostate cancer. J. Clin. Invest. 113, 913–923 (2004)

5. Hermans, K. G. et al. TMPRSS2:ERG fusion by translocation or interstitial deletion is highly relevant in androgen-dependent prostate cancer, but is bypassed in late-stage androgen receptor-negative prostate cancer. Cancer Res. 66, 10658–10663 (2006)

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