Angiogenesis selectively requires the p110α isoform of PI3K to control endothelial cell migration-Reference-Cited by-同舟云学术

Angiogenesis selectively requires the p110α isoform of PI3K to control endothelial cell migration

Published:2008-04-30 Issue:7195 Volume:453 Page:662-666
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Graupera Mariona,Guillermet-Guibert Julie,Foukas Lazaros C.,Phng Li-Kun,Cain Robert J.,Salpekar Ashreena,Pearce Wayne,Meek Stephen,Millan Jaime,Cutillas Pedro R.,Smith Andrew J. H.,Ridley Anne J.,Ruhrberg Christiana,Gerhardt Holger,Vanhaesebroeck Bart

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature06892.pdf

Reference31 articles.

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2. Bi, L. et al. Proliferative defect and embryonic lethality in mice homozygous for a deletion in the p110alpha subunit of phosphoinositide 3-kinase. J. Biol. Chem. 274, 10963–10968 (1999)

3. Jiang, B. H., Zheng, J. Z., Aoki, M. & Vogt, P. K. Phosphatidylinositol 3-kinase signaling mediates angiogenesis and expression of vascular endothelial growth factor in endothelial cells. Proc. Natl Acad. Sci. USA 97, 1749–1753 (2000)

4. Geng, L. et al. A specific antagonist of the p110δ catalytic component of phosphatidylinositol 3′-kinase, IC486068, enhances radiation-induced tumor vascular destruction. Cancer Res. 64, 4893–4899 (2004)

5. Puri, K. D. et al. Mechanisms and implications of phosphoinositide 3-kinaseδ in promoting neutrophil trafficking into inflamed tissue. Blood 103, 3448–3456 (2004)

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