HDAC5 modulates SATB1 transcriptional activity to promote lung adenocarcinoma
Author:
Funder
DST | Science and Engineering Research Board
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Oncology
Link
https://www.nature.com/articles/s41416-023-02341-8.pdf
Reference48 articles.
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2. Ghosh TK, Aparicio-Sanchez JJ, Buxton S, Brook JD. HDAC4 and 5 repression of TBX5 is relieved by protein kinase D1. Sci Rep. 2019;9:17992.
3. Cao C, Vasilatos SN, Bhargava R, Fine JL, Oesterreich S, Davidson NE, et al. Functional interaction of histone deacetylase 5 (HDAC5) and lysine-specific demethylase 1 (LSD1) promotes breast cancer progression. Oncogene. 2017;36:133–45.
4. Liu C, Lv D, Li M, Zhang X, Sun G, Bai Y, et al. Hypermethylation of miRNA-589 promoter leads to upregulation of HDAC5 which promotes malignancy in non-small cell lung cancer. Int J Oncol. 2017;50:2079–90.
5. Hou P, Kapoor A, Zhang Q, Li J, Wu CJ, Li J, et al. Tumor microenvironment remodeling enables bypass of oncogenic KRAS dependency in pancreatic cancer. Cancer Discov. 2020;10:1058–77.
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