AZ304, a novel dual BRAF inhibitor, exerts anti-tumour effects in colorectal cancer independently of BRAF genetic status-Reference-Cited by-同舟云学术

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AZ304, a novel dual BRAF inhibitor, exerts anti-tumour effects in colorectal cancer independently of BRAF genetic status

Published:2018-05 Issue:11 Volume:118 Page:1453-1463
ISSN:0007-0920
Container-title:British Journal of Cancer
language:en
Short-container-title:Br J Cancer

Author:

Ma Rui,Xu Ling,Qu Xiujuan,Che Xiaofang,Zhang Ye,Fan Yibo,Li Ce,Guo Tianshu,Hou Kezuo,Hu Xuejun,Drew Lisa,Shen Minhui,Cheung Tony,Liu Yunpeng

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Oncology

Link

http://www.nature.com/articles/s41416-018-0086-x.pdf

Reference42 articles.

1. Rahman, M. A., Salajegheh, A., Smith, R. A. & Lam, A. K. BRAF inhibitor therapy for melanoma, thyroid and colorectal cancers: development of resistance and further prospects. Curr. Cancer Drug. Targets 14, 128–143 (2014).

2. Mercer, K. E. & Pritchard, C. A. Raf proteins and cancer: B-Raf is identified as a mutational target. Biochim. Biophys. Acta 16539, 25–40 (2013).

3. Di Nicolantonio, F. et al. Wild-type BRAF is required for response to Panitumumab or Cetuximab in metastatic colorectal cancer. J. Clin. Oncol. 26, 5705–5712 (2008).

4. De Roock, W. et al. Effects of KRAS, BRAF, NRAS and PIK3CA mutations on the efficacy of Cetuximab plus chemotherapy in chemotherapy-refractory metastatic colorectal cancer: a retrospective consortium analysis. Lancet Oncol. 11, 753–762 (2010).

5. Pietrantonio, F. et al. Predictive role of BRAF mutations in patients with advanced colorectal cancer receiving Cetuximab and panitumumab: a meta-analysis. Eur. J. Cancer 51, 587–594 (2015).

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