“Paradoxical neoantigenic mutations”: not simply a driver vs passenger dualism
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Oncology
Link
https://www.nature.com/articles/s41416-023-02270-6.pdf
Reference6 articles.
1. Ishino T, Kawashima S, Tanji E, Ueno T, Ueda Y, Ogasawara S, et al. Somatic mutations can induce a noninflamed tumour microenvironment via their original gene functions, despite deriving neoantigens. Br J Cancer. 2023;128:1166–75.
2. Alexandrov LB, Kim J, Haradhvala NJ, Huang MN, Tian Ng AW, Wu Y, et al. The repertoire of mutational signatures in human cancer. Nature 2020;578:94–101.
3. Thornton AM, Fang L, Lo A, McSharry M, Haan D, O’Brien C, et al. eVIP2: Expression-based variant impact phenotyping to predict the function of gene variants. PLoS Comput Biol. 2021;17:e1009132.
4. Boichard A, Pham TV, Yeerna H, Goodman A, Tamayo P, Lippman S, et al. APOBEC-related mutagenesis and neo-peptide hydrophobicity: implications for response to immunotherapy. Oncoimmunology 2019;8:1550341.
5. Litchfield K, Reading JL, Puttick C, Thakkar K, Abbosh C, Bentham R, et al. Meta-analysis of tumor- and T cell-intrinsic mechanisms of sensitization to checkpoint inhibition. Cell 2021;184:596–614.
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