A Pro12Ala substitution in PPARγ2 associated with decreased receptor activity, lower body mass index and improved insulin sensitivity-Reference-Cited by-同舟云学术

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A Pro12Ala substitution in PPARγ2 associated with decreased receptor activity, lower body mass index and improved insulin sensitivity

Published:1998-11 Issue:3 Volume:20 Page:284-287
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

Deeb Samir S.,Fajas Lluis,Nemoto Masami,Pihlajamäki Jussi,Mykkänen Leena,Kuusisto Johanna,Laakso Markku,Fujimoto Wilfred,Auwerx Johan

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng1198_284.pdf

Reference30 articles.

1. Tontonoz, P., Hu, E. & Spiegelman, B.M. Stimulation of adipogenesis in fibroblasts by PPAR γ 2, a lipid-activated transcription factor. Cell 79, 1147–1156 (1994).

2. Tontonoz, P. Hu, E., Graves, R.A., Budavari, A.I. & Spiegelman, B.M. mPPAR γ 2: tissue-specific regulator of an adipocyte enhancer. Genes Dev. 8, 1224– 1234 (1994).

3. Tontonoz, P. & Spiegelman, B.M. Regulation of adipocyte gene expression and differentiation by peroxisome proliferator activated receptor γ. Curr. Opin. Genet. Dev. 5, 571– 576 (1995).

4. Fajas, L. et al. The organization, promoter analysis, and expression of the human PPARγ gene. J. Biol. Chem. 272, 18779–18789 (1997).

5. Werman, A. et al. Ligand-independent activation domain in the N terminus of peroxisome proliferator-activated receptor γ (PPARγ). Differential activity of PARγ 1 and -2 isoforms and influence of insulin. J. Biol. Chem. 272, 20230–20235 (1997).

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