Decreased allergic lung inflammatory cell egression and increased susceptibility to asphyxiation in MMP2-deficiency-Reference-Cited by-同舟云学术

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Decreased allergic lung inflammatory cell egression and increased susceptibility to asphyxiation in MMP2-deficiency

Published:2002-03-11 Issue:4 Volume:3 Page:347-353
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Corry David B.,Rishi Kirtee,Kanellis John,Kiss Attila,Song Li-zhen,Xu Jie,Feng Lili,Werb Zena,Kheradmand Farrah

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/ni773.pdf

Reference48 articles.

1. Barnes, P. J. Pathophysiology of asthma. Br. J. Clin. Pharmacol. 42, 3–10 (1996).

2. Coyle, A. J. et al. Interleukin-4 is required for the induction of lung Th2 mucosal immunity. Am. J. Respir. Cell Mol. Biol. 13, 54–59 (1995).

3. Corry, D. B. et al. Interleukin 4, but not interleukin 5 or eosinophils, is required in a murine model of acute airway hyperreactivity. J. Exp. Med. 183, 109–117 (1996).

4. Foster, P. S., Hogan, S. P., Ramsay, A. J., Matthaei, K. I. & Young, I. G. Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model. J. Exp. Med. 183, 195–201 (1996).

5. Corry, D. B. et al. Requirements for allergen- induced airway hyperreactivity in T and B cell– deficient mice. Mol. Med. 4, 344–355 (1998).

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