α-Catenin acetylation is essential for its stability and blocks its tumor suppressor effects in breast cancer through Yap1
Author:
Funder
National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Molecular Biology,Molecular Medicine
Link
https://www.nature.com/articles/s41417-023-00665-4.pdf
Reference59 articles.
1. Nicholl ID, Matsui T, Weiss TM, Stanley CB, Heller WT, Martel A, et al. α-catenin structure and nanoscale dynamics in solution and in complex with F-actin. Biophys J. 2018;115:642–54.
2. Sun Y, Zhang J, Ma L. α-catenin. A tumor suppressor beyond adherens junctions. Cell Cycle. 2014;13:2334–9.
3. Majewski IJ, Kluijt I, Cats A, Scerri TS, de Jong D, Kluin RJ, et al. An α-E-catenin (CTNNA1) mutation in hereditary diffuse gastric cancer. J Pathol. 2013;229:621–9.
4. Hollestelle A, Elstrodt F, Timmermans M, Sieuwerts AM, Klijn JG, Foekens JA, et al. Four human breast cancer cell lines with biallelic inactivating alpha-catenin gene mutations. Breast Cancer Res Treat. 2010;122:125–33.
5. Siegel RL, Miller KD, Fuchs HE, Jemal A. Cancer statistics, 2022. CA Cancer J Clin. 2022;72:7–33.
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. The Interplay of Acetylation and Ubiquitination Controls PRMT1 Homeostasis;2024-06-18
2. Correction: α-Catenin acetylation is essential for its stability and blocks its tumor suppressor effects in breast cancer through Yap1;Cancer Gene Therapy;2023-09-22
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