Deletion of Pkd1 in renal stromal cells causes defects in the renal stromal compartment and progressive cystogenesis in the kidney
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Molecular Biology,Pathology and Forensic Medicine
Link
http://www.nature.com/articles/labinvest201797.pdf
Reference68 articles.
1. Brasier JL, Henske EP . Loss of the polycystic kidney disease (PKD1) region of chromosome 16p13 in renal cyst cells supports a loss-of-function model for cyst pathogenesis. J Clin Invest 1997;99:194–199.
2. Koptides M, Constantinides R, Kyriakides G et al. Loss of heterozygosity in polycystic kidney disease with a missense mutation in the repeated region of PKD1. Hum Genet 1998;103:709–717.
3. Hopp K, Ward CJ, Hommerding CJ et al. Functional polycystin-1 dosage governs autosomal dominant polycystic kidney disease severity. J Clin Invest 2012;122:4257–4273.
4. Sharif-Naeini R, Folgering JH, Bichet D et al. Polycystin-1 and -2 dosage regulates pressure sensing. Cell 2009;139:587–596.
5. Lantinga-van Leeuwen IS, Dauwerse JG, Baelde HJ et al. Lowering of Pkd1 expression is sufficient to cause polycystic kidney disease. Hum Mol Genet 2004;13:3069–3077.
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