Assembly Pathway of Desmoglein 3 to Desmosomes and Its Perturbation by Pemphigus Vulgaris-IgG in Cultured Keratinocytes, as Revealed by Time-Lapsed Labeling Immunoelectron Microscopy-Reference-Cited by-同舟云学术

Assembly Pathway of Desmoglein 3 to Desmosomes and Its Perturbation by Pemphigus Vulgaris-IgG in Cultured Keratinocytes, as Revealed by Time-Lapsed Labeling Immunoelectron Microscopy

Published:2000-10 Issue:10 Volume:80 Page:1583-1592
ISSN:0023-6837
Container-title:Laboratory Investigation
language:en
Short-container-title:Lab Invest

Author:

Sato Miki,Aoyama Yumi,Kitajima Yasuo

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Link

http://www.nature.com/articles/3780168.pdf

Reference51 articles.

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2. Amagai M, Koch PJ, Nishikawa T, and Stanley JR (1996). Pemphigus vulgaris antigen (Desmoglein 3) is localized in the lower epidermis, the site of blister formation in patients. J Invest Dermatol 106: 351–355.

3. Amagai M (1995). Adhesion molecules. I: Keratinocyte-keratinocytes interaction; cadherins and pemphigus. J Invest Dermatol 104: 146–152.

4. Aoyama Y and Kitajima Y (1999). Pemphigus vulgaris-IgG causes a rapid depletion of desmoglein 3 (Dsg3) from the triton X-100 soluble pools, leading to the formation of Dsg3-depleted desmosomes in a human squamous carcinoma cell line, DJM-1 cells. J Invest Dermatol 112: 67–71.

5. Aoyama Y, Owada MK, and Kitajima Y (1999). A pathogenic autoantibody, pemphigus IgG, induces phosphorylation of desmoglein3, and its dissociation from plakoglobin in cultured keratinocytes. Eur J Immunol 29: 2233–2240.

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